Physiology of Diabetes

Pathophysiologic modification is a change in task as notable from a structural defect. Diabetes occurs when there is a dis-balance between the demand and manufacture of the hormone insulin. Pathophysiology of diabetes is that in this circumstance the immune system outbreaks and terminates the beta cells of the pancreas which produce insulin. There is lack of beta cell leading to complete insulin deficiency. Whereas in type 2 diabetes there is comparatively lesser deficiency of insulin and not an complete deficiency. This means that the body is incapable to harvest appropriate insulin to overcome the demand. There is Beta cell deficiency coupled with peripheral insulin resistanceGestational diabetes is the third type of diabetes. It is caused when there are excessive counter-insulin hormones of pregnancy. This paves the way to a state of insulin resistance and high blood sugar in the mother. There may be faulty insulin receptors. These part of diabetes study is covered in ADA meetings and IDF word Diabetes Congress

  • Physiology of the endocrine pancreas
  • Recent advances in genetics of diabetes
  • Pathogenesis of diabetes
  • Diabetic myelopathy
  • Diabetes and metabolic syndrome
  • Epidemology and pathogenesis of diabetes
  • Pathophysiology of hypoglycemia
  • Physiology of Diabetes Type 2
  • Physiology of Diabetes Type 1
  • Alpha and Beta cells
  • The Role of Incretins in Insulin Secretion

Related Conference of Physiology of Diabetes

July 17-18, 2017

18th European Diabetes Congress

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19thAsia Pacific Diabetes Conference

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9th Annual Congress on Endocrine Disorders and Therapies


(10 Plenary Forums - 1 Event)
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10th International Conference on Endocrinology

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25th Global Diabetes Summit and Medicare Expo

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7th European Diabetes and Endocrinology Congress

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Physiology of Diabetes Conference Speakers

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